A new study conducted by researchers from the University of Michigan and the University of Colorado School of Medicine suggests that ginger supplements may play a critical role in controlling inflammation for individuals living with autoimmune diseases. The study focused on the impact of ginger supplements on a type of white blood cell called neutrophils, specifically on a process known as neutrophil extracellular trap (NET) formation.
NETs are microscopic structures that contribute to inflammation and clotting, which can trigger autoimmune diseases such as lupus, antiphospholipid syndrome, and rheumatoid arthritis. The researchers found that ginger consumption made neutrophils more resistant to NETosis in healthy individuals. This suggests that ginger supplements may help restrain inflammation and symptoms for people with different autoimmune diseases.
In a clinical trial, daily intake of a ginger supplement for seven days resulted in increased levels of a chemical called cAMP inside neutrophils. The high levels of cAMP inhibited NETosis in response to disease-related stimuli. This provides evidence for the biological mechanism underlying ginger’s anti-inflammatory properties in people.
The researchers hope that their findings will encourage healthcare providers and patients to consider ginger supplements as a complementary treatment option for autoimmune and inflammatory diseases, particularly those where neutrophils are overactive. These diseases include lupus, rheumatoid arthritis, antiphospholipid syndrome, and even COVID-19. Further clinical trials are needed to fully understand the potential benefits of ginger in these conditions.
Overall, this study highlights the potential of ginger as a natural supplement to help manage inflammation in autoimmune diseases. It offers a glimpse into the biological mechanism behind ginger’s anti-inflammatory properties and suggests that it could be a valuable addition to treatment programs for individuals with overactive neutrophils.
Source: Melissa Rudy, Fox News Digital (source article not available online)